Because mammalian eggs are produced early in life, while sperm are created continuously, fathers are responsible for a greater share of new mutations passed down to their offspring than mothers. This slightly complicates genetics-based time-since-last-common-ancestor estimations, leading to recent results to the effect that the human-chimp split happened about twice as far back as previously thought. Adam Benton has more information, if you’re interested.
This new paper has prompted Jeffrey Tomkins, the ICR’s go-to geneticist, to publish “Chimp DNA Mutation Study–Selective Yet Surprising.” Tomkins is known for contesting the typically-cited genetic similarity figures of 94-99% and having calculated using his own method a “conservative” (i.e. maximum) figure of nearer 70%.
From the ICR’s URCall series of videos, hosted by Markus Lloyd. (link)
Evolution claims that change occurs from simple creatures to complex creatures – but is that really the case? Since the early 1900’s scientists have been experimenting with fruit flies, to try to produce mutations that result in a major change that evolutionary theory proposes. While over 3000 mutations have been documented, not a single one has resulted in a creature that is anything other than a fruit fly. How long are you willing to wait for science to prove evolution?
You don’t need me to tell you about the upcoming “Ham on Nye” debate – a name with many curious connotations – nor give you my opinion on whether it is a good idea or not. But any aspiring debater needs to be able to almost reflexively parse creationist claims for their most crucial and obvious errors. At present we’re four articles behind, so this is the perfect time to start this series – in future I will probably pull from other, non ICR sources as well.
The format we’ll start off with is to give you a quote from each article to consider, and then (if I can get it to work) you can go to the next page to see what I made of each. Then, head to the comments below to tell me what you would have said. Continue reading →
Attentive viewers will note that this illustration – like Tomkins’ stock photo – is left handed.
Welcome to 2014! I hope you have all had time to settle in a little, and are ready to begin the year afresh. As always, in the event of an earthquake take cover under your desks and then exit through the doors at the front and back in an orderly fashion after the shaking ends. If there is a fire, leave immediately and do not panic. If both occur simultaneously, hope.
Researchers recently studied a highly sophisticated cellular machine that acts as a guard for the genome against harmful mutations and that evolution cannot explain.
Grammar is a wonderful thing – does he mean that “evolution cannot explain” the protein or the mutations? I don’t know for certain, but we’re going to have to use the former as our working assumption. Continue reading →
It’s not an overly interesting “tale” today: Jeffrey Tomkins writes Long Complex Gene Tails Defy Evolution. His topic is a new paper announcing the discovery that mice and humans can both have longer and more numerous 3′ UTRs (messenger RNAuntranslated regions in the 3′ direction relative to the coding sequence – i.e. the magenta section of the above image) than previously thought. The new sequences that have been “conservatively” determined to be of this nature total 6.6 million bases in mice and 5.1 million in humans, which is quite a lot – something along the lines of 0.2% of the size of the entire human genome, though I’m not certain that they can be directly compared. Individually,
they identified 2035 mouse and 1847 human genes that have 3′ UTR tails ranging from 500 to 25,000 bases long. In some cases, they were even longer than the protein-coding areas of the genes themselves.
Tomkins points to the “hundreds to thousands of built in regulatory switches per gene RNA copy.” As you can tell from the title alone he is making a “that’s complex, so it’s out of reach of evolution” argument, which we’ve looked atad nauseam. Given this, there are only two things that are worth clarifying: first, while the individual tails had lengths up to 25,000 bases long the vast majority were much shorter and the average was only a few thousand; second, while Tomkins claims that “[t]hese incredibly long gene tails literally contain hundreds to thousands of genetic switches within each single mRNA,” (emphasis added) the paper only says that “these extensions collectively contain thousands of conserved miRNA binding sites [Tomkins’ “switches”]” (emphasis added). The results are therefore not quite as impressive as they might be, and as Tomkins is selling them.